Apolipoprotein C-III protein concentrations and gene polymorphisms in type 1 diabetes: Associations with lipoprotein subclasses

Publication Description
Serum apolipoprotein C-III (apoCIII) concentration and apoCIII gene polymorphisms have been shown to be a risk factor for cardiovascular disease; however, the underlying mechanisms remain unclear. In addition, no studies have been performed that address these issues in type 1 diabetes. The current study investigated apoCIII protein and apoCIII gene variation in a normotriglyceridemic (82 +/- 57 mg/dL) population of patients with type 1 diabetes, the Diabetes Control and Complications Trial/Epidemiology of Diabetes Intervention and Complications (DCCT/EDIC) cohort. Blood samples were obtained in 409 patients after an overnight fast. Serum apoCIII concentration was highly correlated with multiple changes in lipids and lipoproteins that resulted in an adverse cardiovascular disease risk profile. Higher apoCIII concentrations were associated (P C polymorphism affecting an insulin response element in the apoCIII gene promoter nor a SacI polymorphism in the 3'UTR were associated with any alterations in circulating apoCIII concentrations, serum lipids, apolipoprotein concentrations, lipoprotein composition, or parameters measured by NMR lipoprotein subclass analyses. In summary, elevated apoCIII concentration was associated with risk factors for cardiovascular disease in normolipidemic type 1 diabetic patients through associated changes in lipoprotein subfraction distributions, which were independent of apoCIII genotype.

Primary Author
Klein,Richard L.
McHenry,M. B.
Lok,Kerry H.
Hunter,Steven J.
Le,Ngoc-Anh
Jenkins,Alicia J.
Zheng,Deyi
Semler,Andrea J.
Brown,W. V.
Lyons,Timothy J.
Garvey,W. T.

Volume
53

Issue
10

Start Page
1296

Other Pages
1304

Publisher
Elsevier Inc

URL
https://www.sciencedirect.com/science/article/pii/S0026049504002355 https://www.sciencedirect.com/science/article/pii/S0026049504002355



Reference Type
Journal Article

Periodical Full
Metabolism

Publication Year
2004

Place of Publication
United States

ISSN/ISBN
0026-0495

Document Object Index
10.1016/j.metabol.2004.05.004