The association of previously reported polymorphisms for microvascular complications in a meta-analysis of diabetic retinopathy

Publication Description
We investigated the association of signals from previous GWAS and candidate gene meta-analyses for diabetic retinopathy (DR) or nephropathy (DN), as well as an EPO variant in meta-analyses of severe (SDR) and mild diabetic retinopathy (MDR). Meta-analyses of SDR (≥severe non-proliferative diabetic retinopathy (NPDR) or history of panretinal photocoagulation) and MDR (≥mild NPDR), defined based on seven-field stereoscopic fundus photographs, were performed in two well-characterized type 1 diabetes (T1D) cohorts: the Diabetes Control and Complications Trial/Epidemiology of Diabetes Interventions and Complications (DCCT/EDIC, n = 1,304) and Wisconsin Epidemiologic Study of Diabetic Retinopathy (WESDR, n = 603). Among 34 previous signals for DR, after controlling for multiple testing, no association was replicated in our meta-analyses. rs1571942 and rs12219125 at PLXDC2 locus showed nominally significant (<0.05) association with SDR in the same direction as previous report, as did rs1801282 in PPARG gene with MDR. Among 55 loci previously associated with DN, three showed suggestive associations with SDR in our study without maintaining significance after correction for multiple testing. Of particular interest, rs1617640 (EPO) was not significantly associated with DR status, combined SDR–DN phenotype, time to SDR or time to DN (all P > 0.05). Lack of replication of previous DR hits and EPO despite reasonable statistical power implies that many of these may be false positives. Consistent with pleiotropy, we provide suggestive collective evidence for association between DR and variants previously associated with DN without reaching statistical significance at any single locus.

Primary Author
Hosseini,S. Mohsen
Boright,Andrew P.
Sun,Lei
Canty,Angelo J.
Bull,Shelley B.
Klein,Barbara E. K.
Klein,Ronald
Paterson,Andrew D.

Volume
134

Issue
2

Start Page
247

Other Pages
257

Publisher
Springer Science and Business Media LLC

URL
https://search.datacite.org/works/10.1007/s00439-014-1517-2

PMID
25487307



Reference Type
Journal Article

Periodical Full
Human genetics

Publication Year
2014

Place of Publication
Berlin/Heidelberg

ISSN/ISBN
1432-1203

Document Object Index
10.1007/s00439-014-1517-2