Association of vitamin D status with arterial blood pressure and hypertension risk: a mendelian randomisation study

Publication Description
BACKGROUND: Low plasma 25-hydroxyvitamin D (25[OH]D) concentration is associated with high arterial blood pressure and hypertension risk, but whether this association is causal is unknown. We used a mendelian randomisation approach to test whether 25(OH)D concentration is causally associated with blood pressure and hypertension risk. METHODS: In this mendelian randomisation study, we generated an allele score (25[OH]D synthesis score) based on variants of genes that affect 25(OH)D synthesis or substrate availability (CYP2R1 and DHCR7), which we used as a proxy for 25(OH)D concentration. We meta-analysed data for up to 108 173 individuals from 35 studies in the D-CarDia collaboration to investigate associations between the allele score and blood pressure measurements. We complemented these analyses with previously published summary statistics from the International Consortium on Blood Pressure (ICBP), the Cohorts for Heart and Aging Research in Genomic Epidemiology (CHARGE) consortium, and the Global Blood Pressure Genetics (Global BPGen) consortium. FINDINGS: In phenotypic analyses (up to n=49 363), increased 25(OH)D concentration was associated with decreased systolic blood pressure (beta per 10% increase, -0.12 mm Hg, 95% CI -0.20 to -0.04; p=0.003) and reduced odds of hypertension (odds ratio [OR] 0.98, 95% CI 0.97-0.99; p=0.0003), but not with decreased diastolic blood pressure (beta per 10% increase, -0.02 mm Hg, -0.08 to 0.03; p=0.37). In meta-analyses in which we combined data from D-CarDia and the ICBP (n=146 581, after exclusion of overlapping studies), each 25(OH)D-increasing allele of the synthesis score was associated with a change of -0.10 mm Hg in systolic blood pressure (-0.21 to -0.0001; p=0.0498) and a change of -0.08 mm Hg in diastolic blood pressure (-0.15 to -0.02; p=0.01). When D-CarDia and consortia data for hypertension were meta-analysed together (n=142 255), the synthesis score was associated with a reduced odds of hypertension (OR per allele, 0.98, 0.96-0.99; p=0.001). In instrumental variable analysis, each 10% increase in genetically instrumented 25(OH)D concentration was associated with a change of -0.29 mm Hg in diastolic blood pressure (-0.52 to -0.07; p=0.01), a change of -0.37 mm Hg in systolic blood pressure (-0.73 to 0.003; p=0.052), and an 8.1% decreased odds of hypertension (OR 0.92, 0.87-0.97; p=0.002). INTERPRETATION: Increased plasma concentrations of 25(OH)D might reduce the risk of hypertension. This finding warrants further investigation in an independent, similarly powered study. FUNDING: British Heart Foundation, UK Medical Research Council, and Academy of Finland.

Primary Author
Vimaleswaran,K. S.
Cavadino,A.
Berry,D. J.
LifeLines Cohort Study investigators
Jorde,R.
Dieffenbach,A. K.
Lu,C.
Alves,A. C.
Heerspink,H. J.
Tikkanen,E.
Eriksson,J.
Wong,A.
Mangino,M.
Jablonski,K. A.
Nolte,I. M.
Houston,D. K.
Ahluwalia,T. S.
van der Most, P. J.
Pasko,D.
Zgaga,L.
Thiering,E.
Vitart,V.
Fraser,R. M.
Huffman,J. E.
de Boer,R. A.
Schottker,B.
Saum,K. U.
McCarthy,M. I.
Dupuis,J.
Herzig,K. H.
Sebert,S.
Pouta,A.
Laitinen,J.
Kleber,M. E.
Navis,G.
Lorentzon,M.
Jameson,K.
Arden,N.
Cooper,J. A.
Acharya,J.
Hardy,R.
Raitakari,O.
Ripatti,S.
Billings,L. K.
Lahti,J.
Osmond,C.
Penninx,B. W.
Rejnmark,L.
Lohman,K. K.
Paternoster,L.
Stolk,R. P.
Hernandez,D. G.
Byberg,L.
Hagstrom,E.
Melhus,H.
Ingelsson,E.
Mellstrom,D.
Ljunggren,O.
Tzoulaki,I.
McLachlan,S.
Theodoratou,E.
Tiesler,C. M.
Jula,A.
Navarro,P.
Wright,A. F.
Polasek,O.
International Consortium for Blood Pressure, (ICBP)
Cohorts for Heart and Aging Research in Genomic Epidemiology (CHARGE) consortium
Global Blood Pressure Genetics (Global BPGen) consortium
Caroline Hayward
Wilson,J. F.
Rudan,I.
Salomaa,V.
Heinrich,J.
Campbell,H.
Price,J. F.
Karlsson,M.
Lind,L.
Michaelsson,K.
Bandinelli,S.
Frayling,T. M.
Hartman,C. A.
Sorensen,T. I.
Kritchevsky,S. B.
Langdahl,B. L.
Eriksson,J. G.
Florez,J. C.
Spector,T. D.
Lehtimaki,T.
Kuh,D.
Humphries,S. E.
Cooper,C.
Ohlsson,C.
Marz,W.
de Borst,M. H.
Kumari,M.
Kivimaki,M.
Wang,T. J.
Power,C.
Brenner,H.
Grimnes,G.
van der Harst,P.
Snieder,H.
Hingorani,A. D.
Pilz,S.
Whittaker,J. C.
Jarvelin,M. R.
Hypponen,E.

Author Address
Population, Policy and Practice, UCL Institute of Child Health, London, UK; Hugh Sinclair Unit of Human Nutrition, Department of Food & Nutritional Sciences, School of Chemistry, Food & Pharmacy, University of Reading, Reading, UK.; Population, (TRUNCATED)

Volume
2

Issue
9

Start Page
719

Other Pages
729

Publisher
Vimaleswaran et al. Open Access article distributed under the terms of CC BY. Published by .

Author Address
Population, Policy and Practice, UCL Institute of Child Health, London, UK; Hugh Sinclair Unit of Human Nutrition, Department of Food & Nutritional Sciences, School of Chemistry, Food & Pharmacy, University of Reading, Reading, UK.; Population, (TRUNCATED)

PMID
24974252

PMCID
PMC4582411



Reference Type
Journal Article

Periodical Full
The lancet.Diabetes & endocrinology

Publication Year
2014

Publication Date
Sep

ISSN/ISBN
2213-8595

Document Object Index
S2213-8587(14)70113-5 [pii]