Hyperglucagonemia Does Not Explain the beta-Cell Hyperresponsiveness and Insulin Resistance in Dysglycemic Youth Compared With Adults: Lessons From the RISE Study

Publication Description
OBJECTIVE: To determine whether beta-cell hyperresponsiveness and insulin resistance in youth versus adults in the Restoring Insulin Secretion (RISE) Study are related to increased glucagon release. RESEARCH DESIGN AND METHODS: In 66 youth and 350 adults with impaired glucose tolerance (IGT) or recently diagnosed type 2 diabetes (drug naive), we performed hyperglycemic clamps and oral glucose tolerance tests (OGTTs). From clamps we quantified insulin sensitivity (M/I), plasma fasting glucagon and C-peptide, steady-state glucagon and C-peptide at glucose of 11.1 mmol/L, and arginine-stimulated glucagon (acute glucagon response [AGR]) and C-peptide (ACPRmax) responses at glucose >25 mmol/L. RESULTS: Mean +/- SD fasting glucagon (7.63 +/- 3.47 vs. 8.55 +/- 4.47 pmol/L; P = 0.063) and steady-state glucagon (2.24 +/- 1.46 vs. 2.49 +/- 1.96 pmol/L, P = 0.234) were not different in youth and adults, respectively, while AGR was lower in youth (14.1 +/- 5.2 vs. 16.8 +/- 8.8 pmol/L, P = 0.001). Significant age-group differences in insulin sensitivity, fasting C-peptide, steady-state C-peptide, and ACPRmax were not related to glucagon. Fasting glucose and glucagon were positively correlated in adults (r = 0.133, P = 0.012) and negatively correlated in youth (r = -0.143, P = 0.251). In both age-groups, higher fasting glucagon was associated with higher fasting C-peptide (youth r = 0.209, P = 0.091; adults r = 0.335, P < 0.001) and lower insulin sensitivity (youth r = -0.228, P = 0.066; adults r = -0.324, P < 0.001). With comparable fasting glucagon, youth had greater C-peptide and lower insulin sensitivity. OGTT suppression of glucagon was greater in youth. CONCLUSIONS: Youth with IGT or recently diagnosed type 2 diabetes (drug naive) have hyperresponsive beta-cells and lower insulin sensitivity, but their glucagon concentrations are not increased compared with those in adults. Thus, alpha-cell dysfunction does not appear to explain the difference in beta-cell function and insulin sensitivity in youth versus adults.

Primary Author
Kahn,S. E.
Mather,K. J.
Arslanian,S. A.
Barengolts,E.
Buchanan,T. A.
Caprio,S.
Ehrmann,D. A.
Hannon,T. S.
Marcovina,S.
Nadeau,K. J.
Utzschneider,K. M.
Xiang,A. H.
Edelstein,S. L.
RISE Consortium
Rise Consortium Investigators:

Author Address
VA Puget Sound Health Care System, Seattle, WA.; University of Washington, Seattle, WA.; Indiana University School of Medicine, Indianapolis, IN.; UPMC Children's Hospital of Pittsburgh, Pittsburgh, PA.; Jesse Brown VA Medical Center, Chicago, I(TRUNCATED)

Volume
44

Issue
9

Start Page
1961

Other Pages
1969

Publisher
by the American Diabetes Association

Author Address
VA Puget Sound Health Care System, Seattle, WA.; University of Washington, Seattle, WA.; Indiana University School of Medicine, Indianapolis, IN.; UPMC Children's Hospital of Pittsburgh, Pittsburgh, PA.; Jesse Brown VA Medical Center, Chicago, I(TRUNCATED)

PMID
34131047

PMCID
PMC8740916



Reference Type
Journal Article

Periodical Full
Diabetes care

Publication Year
2021

Publication Date
1-Sep

Place of Publication
United States

ISSN/ISBN
1935-5548

Document Object Index
10.2337/dc21-0460 [doi]

Accession Number
PMID: 34131047